Apolipoprotein E Deficiency Aggravates Neuronal Injury by Enhancing Neuroinflammation via the JNK/c-Jun Pathway in the Early Phase of Experimental Subarachnoid Hemorrhage in Mice

<div>The schema of in vivo study design. SAH: subarachnoid hemorrhage; TUNEL: terminal-deoxynucleoitidyl transferase-mediated nick end labeling; IHC: immunohistochemistry; WB: western blot; Clod. Lip.: clodronate liposome; I.C.V.: intracerebroventricularly.</div>

Oxidative Medicine and Cellular Longevity

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Figure 1

Figure 1: Apolipoprotein E Deficiency Aggravates Neuronal Injury by Enhancing Neuroinflammation via the JNK/c-Jun Pathway in the Early Phase of Experimental Subarachnoid Hemorrhage in Mice 

Figure 1 | Apolipoprotein E Deficiency Aggravates Neuronal Injury by Enhancing Neuroinflammation via the JNK/c-Jun Pathway in the Early Phase of Experimental Subarachnoid Hemorrhage in Mice 