The Ability of Different Ketohexoses to Alter Apo-A-I Structure and Function In Vitro and to Induce Hepatosteatosis, Oxidative Stress, and Impaired Plasma Lipid Profile in Hyperlipidemic Zebrafish

<div>Change in embryo stage after coinjection of oxLDL and ketohexose-treated apoA-I at 0 and 25 hr postinjection. In order to compare production of reactive oxygen species, red fluorescence image (Ex = 588 nm, Em = 605 nm) of zebrafish embryo was obtained using a Nikon Eclipse TE2000 microscope (Tokyo, Japan) after dihydroethidium (DHE) staining as described in the text. f-A-I: fructose-treated apoA-I; t-A-I: tagatose-treated apoA-I; p-A-I: psicose-treated apoA-I; H<sub>2</sub>O-A-I: H<sub>2</sub>O-treated apoA-I.</div>

Oxidative Medicine and Cellular Longevity

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Figure 8

Figure 8: The Ability of Different Ketohexoses to Alter Apo-A-I Structure and Function In Vitro and to Induce Hepatosteatosis, Oxidative Stress, and Impaired Plasma Lipid Profile in Hyperlipidemic Zebrafish 

Figure 8 | The Ability of Different Ketohexoses to Alter Apo-A-I Structure and Function In Vitro and to Induce Hepatosteatosis, Oxidative Stress, and Impaired Plasma Lipid Profile in Hyperlipidemic Zebrafish 