The Ability of Different Ketohexoses to Alter Apo-A-I Structure and Function In Vitro and to Induce Hepatosteatosis, Oxidative Stress, and Impaired Plasma Lipid Profile in Hyperlipidemic Zebrafish

<div>Western blot analysis. An equal amount of protein (13 <i>μ</i>g per lane) was loaded on 15% SDS-PAGE. ApoA-I (ab7613; Abcam), tumor necrosis factor (TNF)-<i>α</i> (sc52746; Santa cruz biotechnology), and <i>β</i>-actin antibodies (ab8229; Abcam) were used.</div>

Oxidative Medicine and Cellular Longevity

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Figure 6

Figure 6: The Ability of Different Ketohexoses to Alter Apo-A-I Structure and Function In Vitro and to Induce Hepatosteatosis, Oxidative Stress, and Impaired Plasma Lipid Profile in Hyperlipidemic Zebrafish 

Figure 6 | The Ability of Different Ketohexoses to Alter Apo-A-I Structure and Function In Vitro and to Induce Hepatosteatosis, Oxidative Stress, and Impaired Plasma Lipid Profile in Hyperlipidemic Zebrafish 