The Ability of Different Ketohexoses to Alter Apo-A-I Structure and Function In Vitro and to Induce Hepatosteatosis, Oxidative Stress, and Impaired Plasma Lipid Profile in Hyperlipidemic Zebrafish

<div>Antioxidant ability of apoA-I, which was treated by each ketohexose, against oxidation of LDL. (a) Detection of conjugated diene midst copper-mediated LDL oxidation. Continuous monitoring of conjugate diene level at absorbance 234 nm (A<sub>234</sub>) during copper-mediated oxidation in the presence of either rHDL containing native apoA-I or ketohexose treated apoA-I. (b) Electromobility of LDL, which was oxidized by Cu<sup>2+</sup> under presence of ketohexose treated apoA-I-rHDL, on 0.5% agarose gel.</div>

Oxidative Medicine and Cellular Longevity

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Figure 3: The Ability of Different Ketohexoses to Alter Apo-A-I Structure and Function In Vitro and to Induce Hepatosteatosis, Oxidative Stress, and Impaired Plasma Lipid Profile in Hyperlipidemic Zebrafish 

Figure 3 | The Ability of Different Ketohexoses to Alter Apo-A-I Structure and Function In Vitro and to Induce Hepatosteatosis, Oxidative Stress, and Impaired Plasma Lipid Profile in Hyperlipidemic Zebrafish 