ceAF Ameliorates Diabetic Wound Healing by Alleviating Inflammation and Oxidative Stress via TLR4/NF-κB and Nrf2 Pathways

Figure 6

ceAF was able to suppress inflammation via promoting M2 macrophage polarization in vivo. (a) The expression of CD206, Arg-1, iNOS, IL-1β, IL-6, and TNF-α in the diabetic wound of mice was evaluated by WB. GAPDH served as a standard reference. (b) WB blots in (a) were quantified by densitometric analysis. (c–h) The expression of CD206, Arg-1, iNOS, IL-1β, IL-6, and TNF-α in the diabetic wound of mice was tested by RT-qPCR. , . , , .

(a)

(b)

(c)

(d)

(e)

(f)

(g)

(h)