Effect of N-Acetyl Cysteine on Intracerebroventricular Colchicine Induced Cognitive Deficits, Beta Amyloid Pathology, and Glial Cells

<p>Photomicrographs of the hippocampal subregions in different groups of rats immunostained for <i>β</i>-amyloid protein (BAP). Note the expression of BAP (arrow) in large number of neurons in AD group in all regions. The number of BAP positive neurons was less or not present in NAC treated groups (AD+NAC 100) (photomicrographs of sham and AD+NAC 50 groups were avoided for simplicity). Scale bar= 25<i>μ</i>, in CA1-CA4 regions, =15<i>μ</i> in dentate gyrus (DG).</p>

Neuroscience Journal

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Figure 2

Figure 2: Effect of N-Acetyl Cysteine on Intracerebroventricular Colchicine Induced Cognitive Deficits, Beta Amyloid Pathology, and Glial Cells 