Lipid Bodies: Inflammatory Organelles Implicated in Host-Trypanosoma cruzi Interplay during Innate Immune Responses
Figure 5
Trypanosoma cruzi infection induces concomitant lipid body formation and prostaglandin E2 (PGE2) synthesis. Associations between number of lipid bodies (bars) and prostaglandin E2 (PGE2) peritoneal levels (line) in rats at day 6 or 12 of infection with T. cruzi and in uninfected controls. At both days, the lipid body numbers were significantly increased () in parallel to an accentuated increase of PGE2 synthesis. Data are expressed as means ± SEM. Reprinted from [11] with permission.
