Relevance of a Hypersaline Sodium-Rich Naturally Sparkling Mineral Water to the Protection against Metabolic Syndrome Induction in Fructose-Fed Sprague-Dawley Rats: A Biochemical, Metabolic, and Redox Approach

Figure 1

(a) Body weight (g; 𝑛=7; *𝑃<0.05 CONT versus FRUCTMIN (Δ body weight (g; 𝑛=7), between weeks 8 and 0 in the inset; ^#𝑃<0.01 FRUCT and FRUCTMIN versus CONT)), (b) food ingestion (g; 𝑛=7; *𝑃<0.05 CONT versus FRUCTMIN, and (c) fluid ingestion evolution (mL; 𝑛=7; ⁺𝑃<0.05 CONT versus FRUCT and FRUCTMIN; ^#𝑃<0.001 CONT versus FRUCT and FRUCTMIN; ^$𝑃<0.05 CONT versus FRUCT and 𝑃<0.01 CONT versus FRUCTMIN; ^$$𝑃<0.01 CONT versus FRUCT and 𝑃<0.05 CONT versus FRUCTMIN; **𝑃<0.01 CONT versus FRUCTMIN), during the dietary intervention. Results were expressed as mean ± standard error of the mean. CONT: control; FRUCT: 10% fructose in tap water; FRUCTMIN: 10% fructose in natural mineral-rich water.

(a)

(b)

(c)