Accelerated Oxalosis Contributing to Delayed Graft Function after Renal Transplantation

Figure 1

(a) H&E stain showing extensive acute tubular necrosis with associated peritubular capillaritis and interstitial nephritis; (b) H&E stain showing polarizable oxalate crystals in a tubule; (c) H&E stain showing active endothelialitis in one large caliber artery, (d) electron microscopy showing electron-dense deposits with immunoreactivity for IgA, in keeping with a remote IgA-dominant immune complex mediated nephropathy in the donor.

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(b)

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